Curcumin , a JNK blocker , blocked ATRA inhibition of cell growth.
atra对 Y79 细胞的抑制作用可被JNK的 阻断剂Curcumin阻断.
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The TM transcription and expression upregulated by ATRA may alleviate dysfunction of coagulation in APL.
ATRA增高TM转录及表达,缓解APL患者凝血功能异常.
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Curcumin , a JNK blocker, blocked the apoptosis and the growth inhibition induced by ATRA.
ATRA可诱导Y79细胞凋亡,此凋亡过程可被JNK的 阻断剂Curcumin阻断.
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Objective : To detect the mechanism of hyperleukocytosis in patients with APL after treatment with ATRA.
目的: 揭示APL患者ATRA治疗后高白细胞征的机制.
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IntroductionAll - trans retinoic acid ( ATRA ) is efficiently differentiation in - ducer for acute promyelocytic leukemia ( APL ).
全反式维甲酸 ( ATRA ) 用于急性早幼粒细胞白血病 ( APL) 的诱导分化治疗,有很高的完全缓解(CR ) 率,现已成为APL诱导缓解治疗的首选药物.
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Curcumin, JNK blocker , blocked the growth inhibition by ATRA.
此生长抑制过程可被 JNK 的阻断剂Curcumin阻断.
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Conclusion There are significant improvement in prognosis of APL treated with ATRA.
结论全反式维甲酸对急性早幼粒细胞性白血病患者的预后有很大改善.
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Objective To observe the clinical effects of all - trans - retinoic acid ( ATRA ) in treating 11 patients with acute promyelocytic leukemia ( APL ).
目的观察11例急性早幼粒细胞性白血病患者经全反式 维 甲酸 治疗后的临床效果.
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Objective To determine the effect of all - trans retinoic acid ( ATRA ) on human tongue carcinoma.
目的研究维 甲酸 对舌癌细胞株的诱导分化作用.
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In contrast, APL cells of two cases showed synergistic differentiation after treatment with ATAR and bufalin.
为了进一步研究蟾蜍灵对ATRA诱导分化的增强效应, 我们研究蟾蜍灵与维甲酸联合对APL原 代培养细胞的诱导分化作用.
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