Methods: 33 post - thoracotomy patients were divided into DCA, PCEA and PCIA groups.
方法: 将开胸术后病人33例随机平均分入DCA组 、 PCEA组、PCIA组.
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DCA up - regulated the expression of AP - l ( c - Jun c - Fos ) and COX - 2 in a dose dependent manner.
DCA能上调AP -1 ( c -Jun 、c-Fos ) 蛋白及COX -2 蛋白表达,呈浓度依赖性.
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In this case, LSS - DCA aligned the sequences with the same way as ClustalW.
在这种情况下, LSS-DCA的策略就不起作用,只采用ClustalW对齐序列.
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The kinetics and mechanism of exciplex formation of 9,10 - dicyanoanthracene ( DCA ) by acenaphthenone ( ANO ) have been studied.
研究了苊酮 ( ANO ) 对9,10-二氰蒽 ( DCA ) 的荧光猝灭与激基络合物形成的动力学与机理.
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Results: 200 μ M DCA induced cell proliferation and COX - 2 mRNA transcription.
结果: 200μMDCA可以 促进HT-29 细胞的增殖,并诱导COX -2 mRNA的转录、COX-2蛋白的表达及PGE2的合成.
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