Mycosporine - like amino acids ( MAAs ) strongly absorb UVR and protect cells against harmful UVR.
类菌孢素氨基酸(mycosporine-like aminoacids,间称MAAs ) 存在于许多生物体内,对紫外辐射 ( UVR) 有较高的吸收能力,被认为有保护细胞、减少UVR损伤的功能.
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Objective To investigate the protective effect of nicotinamide on UVR - induced DNA damage.
目的探讨烟酰胺对紫外线照射诱发DNA损伤的保护作用.
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Thus, risk evaluation of cataract induced by UVR is an urgent need.
因此, 亟待开展紫外线与白内障的危险性评价.
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There was a 9.97 % ～67.1 % decrease in the population of LC after UVR.
表皮内LC密度降低,细胞数较照射前减少9.7%～67.01%.
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The density and structure of epidermal LC returned to normal 8 days after cessation of UVR.
停止照射后8天LC密度及结构基本复原.
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Conclusion nicotinamide is a perfect antagonist to UVR - induced DNA damage within fitting concentration.
结论适宜浓度的烟酰胺是抗紫外线照射诱发DNA损伤的拮 抗 剂.
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UVR and chemicals are two major regulating factors which affecting the melanin genesis or melanin transport.
虽然影响体内黑素合成及运输的因素繁多,黑素合成机制也较为复杂,但紫外线和化学物质是影响和调控肤色的两大主要因素.
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Objective To investigate the relationship between personal exposure of ultraviolet radiation and ambient solar UVR.
目的 了解个体紫外线暴露剂量与环境紫外线剂量的关系.
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