Our Iaboratory had researched different degree stress influenceon aging, and discovered that proper stress stimulus (swimmingl5min in l8C water every day) could delay mice aging processinduced by D-galactose.
本实验室前阶段工作研究了不同强度的应激对衰老的影响，发现适宜的应激刺激（水温18℃，每日游泳15min）可以逆转由D-半乳糖诱导的小鼠衰老进程，并研究表明这种适宜应激抗衰老的作用与自由基机制有关。